Chap 32

watery fluid (CHF, Live disease, kidney disorder) that decrease proteins, decreases plasma oncotic pressure

puss like

less watery increased concentrations of WBC and plasma proteins occurs in resp to inflammation, infection or CA

mucous

pneumothorax

air or gas in pleural space

destruction of alveoli breakdown of elastin ~ smoking, inhaled toxins, V/Q mismatch, hypoxemia, airtrapping

Fx of several consecutive ribs or fx of sternum

exchange of O2/Co2

mechanical movement of gas or air in/out of lungs

capillary blood flow high to low

amount of blood flow into systemic capillaries normal V/Q 0.8 lungs are better perfused than ventilated

smooth MM of airway sensitive to increase in size of lungs decreased vent rate/volume with increased stimulation

monitors arterial blood by sensing increased in Ph of CSF

baroreceptors

detect PaCO2 and amount of O2 in arterial blood (PaO2)

C-Section

natural birth

infants of mother with DM

Premie

full-term

immature lungs

destruction of alveoli breakdown of elastin ~ smoking, inhaled toxins, V/Q mismatch, hypoxemia, airtrapping

air or gas in pleural space destroying negative pressure

fx several consecutive ribs or fx sternum and ribs

air trapping

Left sided HF, increase in thickening of vessel wall with luminal narrowing abnormal vasoconstriction increased resistance to pulmonary artery blood flow

pneumthorax

PE

inadequate gas exchange

abnormal sputum

pain

able to speak in a normal tone and clarity

abnormal breathing

air hunger

RR 12-20

bronchio dilation

inadequate gas exchange, hypoxemia direct or indirect injury

chronic interaction of airway obstruction, bronchial hyperresponsiveness and inflammation, mast cells, eosinophils, neuts, T Lymph, macrophages, and damaged epithelial cells typical dx in childhood or after 40 years

abnormal secretions

stiffening of chest wall

increased respiratory strength and endurance

decreased risk for infection

loss of elastic recoil

altered gas exchange

increased ability to compensate

abnormal secretions causing obstructive problems "mucous plugging"

acute resp failure onset

bronchio dilation

chronic interaction of airway obstruction, bronchial hyperresponsiveness and inflammation, mast cells, eosinophils, neuts, T Lymph, macrophages, and damaged epithelial cells typical dx in childhood or after 40 years

amount of blood flow into systemic capillaries normal V/Q 0.8 lungs are better perfused than ventilated

capillary blood flow from high to low

exchange of O2 and Co2 during cell metabolism

mechanical movement of gas or air in/out of lungs

chronic interaction of airway obstruction, bronchial hyperresponsiveness and inflammation, mast cells, eosinophils, neuts, T Lymph, macrophages, and damaged epithelial cells typical dx in childhood or after 40 years

inadequate gas exchange

abnormal secretions "mucous plugging"

Defined as resp failure acute onset, with severe hypoxemia, bilateral infiltrates on CXR without evidence of HF

mechanical movement of gas or air in/out of lungs

exchange of gas during cell metabolism

capillary blood flow from high to low

amount of blood flow into systemic capillaries normal V/Q 0.8 lungs are better perfused than ventilated

increases atelectasis

coats alveolous facilitates the expansion with inspiration and decreases surface tension preventing lungs from collapsing

increases surface tension

contracts

decreased ability to expand

increases ability of lungs to expand

increased fluid

impairs ventilation

difficulty breathing

enhances ventilation

occlusion of a portion of pulmonary vascular

Death

BLE calf pain, tenderness

symmetric calfs

drinking

occupational

cigarette smoking

hereditary

watery fluid (CHF, Live disease, kidney disorder) that decrease proteins, decreases plasma oncotic pressure

puss like

mucous

less watery increased concentrations of WBC plus plasma proteins occurs in resp to inflammations, infection, or CA

pain receptors

detect PaCO2 and amount of O2 in arterial blood (PaO2)

mechanical receptors

baroreceptors

mechanical receptors

baro receptors

monitor arterial blood by sensing increased in Ph of CSF near respiratory center

detect PaCO2 and amount of O2 in arterial blood (PaO2)

disease detected usually at 25 years of age

chronic interaction of airway obstruction, bronchial hyperresponsiveness and inflammation, mast cells, eosinophils, neuts, T Lymph, macrophages, and damaged epithelial cells typical dx in childhood or after 40 years

non obstructive airway disease

bronchial dilation

mechanical movement of gas or air in/out of lungs

amount of blood flow into systemic capillaries normal V/Q 0.8 lungs are better perfused than ventilated

capillary blood flow from high to low

lipoprotein that coats alveolous

air or gas in pleural space

Fx several consecutive ribs or fx sternum plus ribs

pneumothorax

destruction of alveoli breakdown of elastin ~ smoking, inhaled toxins, V/Q mismatch, hypoxemia, airtrapping