OKEP 10th June'16

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The H.pyroli infection causes__________,________ & _____________.

a. chronic inflammation of all or part of your digestive & respiratory tract

b. chronic inflammation of all or part of your digestive tract

c. chronic gastritis, gastric and gastric adenoid carcinoma

d. Chronic cancer of small intestine

H. pylori effects on oxidative stress in chronic gastritis patients showed that the amount of______ and were ________increased in patients.

a. Malondialdehyde, reduced glutathione

b. MDH, Methionine

c. GSSH, GSH

d. Malondialdehyde, oxidized glutathione

Depletion of________ may result in the accumulation of free radicals that can initiate membrane damage by lipid peroxidation.

a. Gastric mucosal GSSH

b. GST

c. X

d. Gastric mucosal GSH

H. pylori directly _________.

a. decrease cellular glutathione

b. increases GST level that causes damage

c. reduces gastrin level

d. Both A & C

A decreased level of _______has been detected in the gastric mucosa of patients with H. pylori infection.

a. GSSH:GSH

b. GSH

c. GST

d. GPx

___________in patients with gastritis as a result of H. pylori were significantly increased as compared to the control groups.

a. Lipoxygenease

b. NADPH

c. Malondialdehyde (MDA)

d. MDA & NADPH

10.

Reduced glutathione & TAC were found to be increased in H pyroli patients.

True

False

11.

Depletion of________ may result in the accumulation of free radicals that can initiate membrane damage by lipid peroxidation.

a. GSSH

b. Gastric mucosal GSH

c. Mucosal content of CSF

d. None of the above

12.

Which of the following is not mentioned indication in Thiotres LBL?

a. Alcoholic Liver Disease

b. Liver Cirrhosis

c. Liver Damage in HIV/HCV infection

d. In Gastritis

e. Skin whitening agent

13.

Glutathione in Thiotres comes in ______ form.

Oxidized form

Reduced form

Basic form

Acidic form

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