Thrombosis Pathology Exam 2

1.

1 point

Stage of shock where cellular and tissue damage is severe and survival is not possible

Irreversible stage

Progressive stage

Get off the stage

Nonprogressive stage

2.

1 point

Break down of fibrin by plasmin to modulate clot size; results in fibrin split products such as "d-dimers"

Primary hemostasis

Secondary hemostasis

Fibrinolysis

Vasoconstriction

3.

1 point

Release of endothelin at site of injury temporarily preventing loss of blood

Fibrinolysis

Secondary hemostasis

Primary hemostasis

Vasoconstriction

4.

1 point

Loss of vascular tone due to anesthesia or spinal cord injury

Cardiogenic shock

anaphylactic shock

neurogenic shock

hypovolemic shock

septic shock

5.

1 point

A mural thrombus is formed in:

Deep veins of the legs

Bone marrow

Arteries only

heart or aortic lumen

6.

1 point

Most frequently caused by IV or central lines

Fat embolism

systemic thromboembolism

Air embolism

Amniotic fluid embolism

Pulmonary Thromboembolism

7.

1 point

When a thrombus is removed by fibrinolytic cascade

Dissolution

Propagation

Recanalization

Embolism

8.

1 point

Venous thrombi grow retrograde and arterial thrombi grow in direction of blood flow.

True

False

9.

1 point

Results from blood pooling in response to microbial infection

hypovolemic shock

anaphylactic shock

septic shock

neurogenic shock

Cardiogenic shock

10.

1 point

An example of an infarct would be:

DVT

area of dead tissue caused by occlusion

occlusion of pulmonary arteries

Fat embolism

atheroma

11.

1 point

tissue factors are released and coagulation factors form thrombin which converts fibrinogen to fibrin and stabilizes "plug"

Vasoconstriction

Primary hemostasis

Secondary hemostasis

Fibrinolysis

12.

1 point

Caused by loss of intravascular pressure frequently from fluid loss or burns

Cardiogenic shock

septic shock

neurogenic shock

anaphylactic shock

hypovolemic shock

13.

1 point

Intravascular solid, liquid, or gas that lodges in a vessel resulting in occlusion

Recanalization

Embolism

Dissolution

Propagation

14.

1 point

Result from soft tissue crush injury or long bone fracture

Air embolism

Amniotic fluid embolism

systemic thromboembolism

Pulmonary Thromboembolism

Fat embolism

15.

1 point

Not a common cause of left sided heart failure

Pulmonary Embolism

IHD (Ischemic Heart Disease)

Aortic/Mitral disease

HTN (Systemic Hypertension)

16.

1 point

Causes sudden hypoxia and frequently occurs when DVT fragments:

systemic thromboembolism

Pulmonary Thromboembolism

Fat embolism

Air embolism

Amniotic fluid embolism

17.

1 point

Describes the stage of shock in which compensatory mechanisms are activated and perfusion of vital organs is maintained

Get off the stage

Nonprogressive stage

Irreversible stage

Progressive stage

18.

1 point

Which of the following is not a characteristic of an arterial thrombi?

Grow retrograde from point of attachment

Develop at endothelial injuries

major cause of nontraumatic limb amputation

directly responsible for varicosities

19.

1 point

Stage of shock during which decompensation begins and tissue hypoperfusion leads to hypoxia and acidosis

Nonprogressive stage

Irreversible stage

Progressive stage

Get off the stage

20.

1 point

Not a factor when it comes to development of infarction

Source of thrombus

Vulnerability to hypoxia

Elevated oxygen content in blood

Rate of development of occlusion

Nature of vascular supply

21.

1 point

Definition of "shock"

Seeing your grade on your #2 path exam

spasm of systemic vasculature

massive, intraperitoneal infection

Systemic hypoperfusion

22.

1 point

Continual growth of a thrombus until it breaks or "embolizes"

Recanalization

Embolism

Propagation

Dissolution

23.

1 point

Saphenous vein is a deep vein and a common site for DVT

definitely

Nope

ya sure

Not too sure...

24.

1 point

IgE mediated hypersensitivity results in systemic vasodilation

anaphylactic shock

Cardiogenic shock

hypovolemic shock

septic shock

neurogenic shock

25.

1 point

Caused by "pump" failure

hypovolemic shock

anaphylactic shock

Cardiogenic shock

neurogenic shock

septic shock

26.

1 point

Which of these is NOT one of the primary abnormalities of Virchow's (Weercoff's) Triad that leads to thrombus formation?

Endothelial Injury

Hypercoagulability

Hyperbilirubinemia

Abnormal Blood Flow

27.

1 point

Arise from cardiac mural thrombi (dilated left atria/ventricle) or aortic aneurysms

Fat embolism

systemic thromboembolism

Amniotic fluid embolism

Air embolism

Pulmonary Thromboembolism

28.

1 point

Growth of capillary channels around a thrombus in an attempt to reestablish flow into the original vascular lumen

Dissolution

Propagation

Recanalization

Embolism

29.

1 point

90% of venous blood volume is in deep veins vs 10% in the superficial veins

True

False

30.

1 point

platelet response once exposed to subendothelial matrix and forms a hemostatic plug

Primary hemostasis

Vasoconstriction

Secondary hemostasis

Fibrinolysis

31.

1 point

Rare with mortality as high as 40% and occurs during childbirth

Pulmonary Thromboembolism

Air embolism

Fat embolism

Amniotic fluid embolism

systemic thromboembolism

32.

1 point

An important clinical significance of venous thrombosis is:

Can cause dissection

can cause varicosities

result in emboli that lodge in the pulmonary arteries

leads to MI